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INVESTIGATIVE
UROLOGY
Intravesical
nitric oxide production discriminates between classic and nonulcer interstitial
cystitis
Logadottir Y, Ehren I, Fall M, Wiklund NP, Peeker R, Hanno PM
From the Department of Urology, Sahlgrenska University Hospital, Göteborg
(YL, MF, RP), and Department of Urology, Karolinska University Hospital,
Stockholm (IE, NPW), Sweden
J Urol. 2004; 171:1148-51
- Purpose:
Interstitial cystitis (IC) is one of the most bothersome conditions
in urological practice. There are 2 subtypes, classic and nonulcer IC,
with similar symptoms but different outcomes with respect to clinical
course and response to treatment. Histologically there are fundamental
differences between the 2 subtypes, classic IC presenting a severe abnormality
of the urothelium and characteristic inflammatory cell infiltrates while
inflammation is scant in nonulcer IC. Regulation of urinary nitric oxide
synthase activity has been proposed to be of importance for immunological
responses in IC. We present evidence of a profound difference between
the 2 subtypes concerning nitric oxide production, mirroring the differences
in inflammatory response in IC.
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Materials and Methods:
A total of 17 patients with both subtypes and active disease as well
as patients with disease in remission were included in the study, all
diagnosed according to National Institute for Diabetes and Digestive
and Kidney Diseases criteria. Luminal nitric oxide was measured in the
bladder of patients using a chemiluminescence nitric oxide analyzer.
- Results:
All patients with classic IC had high levels of NO. None of
the other patients had any significant increase in NO levels in the
bladder. The NO level in patients with classic IC was not related to
symptoms but rather to the assignment to this specific subgroup of IC.
The highest levels of NO were found in patients in the initial phase
of classic IC.
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Conclusions:
The difference in NO evaporation between classic and nonulcer IC allows
for subtyping of cases meeting National Institute for Diabetes and Digestive
and Kidney Diseases criteria without performing cystoscopy. The findings
in the present series together with previous findings clearly demonstrate
that the 2 subtypes of IC represent separate entities. This separation
further emphasizes the need to subtype all cases included in all scientific
matters, ensuring that the 2 subtypes are evaluated separately in clinical
studies.
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Editorial Comment
Interstitial cystitis (IC) is often subdivided into 2 different subtypes:
the classic “ulcerous” form of interstitial cystitis and
the “early” or “nonulcer” form. The differences
between the 2 subtypes are reflected in clinical manifestation and age
distribution. It has also been demonstrated that the 2 subtypes respond
differently to many treatment procedures (1). The main tool for differential
diagnosis between the 2 forms of disease has been cystoscopy.
Classic IC presents at endoscopy with reddened mucosal areas. These
are often associated with small vessels radiating towards a central
scar that ruptures with increasing bladder distension. Histological
specimens obtained from lesions demonstrate that classic IC is a destructive
inflammation and some patients eventually develop a small capacity fibrotic
bladder. Outflow obstruction of the upper urinary tract may also occur
in the final stage of classic IC.
In nonulcer IC, the bladder mucosa is normal at initial cystoscopy.
The development of glomerulations after hydrodistension is considered
to be a positive diagnostic sign. Histologically, there are no or scant
inflammatory signs in nonulcer disease (1).
In the present pioneer study, the authors demonstrated that all patients
with classic IC showed high or very high levels of NO. None of the other
patients had any significant increase in NO in the bladder. The NO level
in patients with classic IC was not related to symptoms but rather to
the assignment to this specific subgroup of IC. However, disease stage
seemed to influence NO levels with the highest levels of NO found in
patients in the initial phase of classic IC. The difference in NO levels
between classic and nonulcer IC allows for subtyping of cases without
performing cystoscopy.
Reference
1. Peeker R, Fall M: The impact of heterogeneity on the diagnosis and
treatment of interstitial cystitis. Int Braz J Urol. 2002; 28: 10-19.
Dr.
Francisco J.B. Sampaio
Full-Professor and Chair, Urogenital Research Unit
State University of Rio de Janeiro
Rio de Janeiro, Brazil
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