|
UROGENITAL
TRAUMA
Self-expanding
metallic stent placement for renal artery dissection due to blunt trauma
Inoue S, Koizumi J, Iino M, Seki T, Inokuchi S
Department of Emergency Medicine, Tokai University School of Medicine,
Isehara
City, Kanagawa, Japan
J Urol. 2004; 171: 347-8
- Case
Report:
No abstract available
- Editorial
Comment
Reports in the literature concerning the successful treatment of blunt
renal artery injury with endovascular methods are rare (3 cases in the
literature). Endovascular treatments are very tempting, because open
repair can be both dangerous and futile, with a high rate of post-surgical
thrombosis. Also, most patients with open arterial repairs would be
treated with anticoagulants to decrease the potential for postoperative
thrombosis, although this is often not possible in a trauma population.
The authors of this case report discuss a patient with a traumatic intimal
tear of the renal artery which caused both renal hypoperfusion and renovascular
hypertension, who was treated with placement of a wallstent in the artery.
Renal perfusion improved immediately and the hypertension subsided.
The authors gave heparin 10,000 IU for 48 hours followed by aspirin
and the phosphodiesterase III inhibitor (cilostazol) for 3 months. The
patient suffered no bleeding, which was surprising as she had liver
and bilateral lung contusions. Although these authors show that endovascular
treatment of significant traumatic renal artery stenosis is possible
I believe that (although tempting) it likely remains impractical for
the majority of out trauma patients whom we are unwilling to fully anticoagulate
after their injury. Interventional radiology physicians also remain
wary of placing stents in injured vessels because of the concern of
artery rupture or stent migration, causing catastrophic bleeding (although
these authors advocate both endoluminal ultrasound and the use of a
long stent to make sure the entire injured portion is stented properly).
Perhaps the future will bring an endoluminal arterial stent technology
that won’t require systemic anticoagulation. Until then, this
potentially risky treatment will remain experimental at best.
Dr.
Richard A. Santucci
Assistant Professor of Urology
Wayne State University
Detroit, Michigan, USA
|